Symptoms of GS are not clearly identified in elevated levels of bilirubin. Most of the people don’t come to a realisation of having it, because the bilirubin levels mostly stay within normal limits and rarely rise – that too slightly, to an extent that is tolerable. However, substances that are glucuronidated by the affected enzymes could cause symptoms such as: Fatigue Difficulty in concentration Anxiety Loss of appetite Abdominal pain and itching without rashesDFDAFF
The only cause of Gilbert’s Syndrome is an abnormal gene from either parent. It affects the liver and gives birth to an excessive amount of bilirubin. A gene in the liver controls enzyme which breaks down the bilirubin. A person with an abnormal gene stores a large amount of bilirubin. The syndrome is caused by: Lack or deficiency of the uridine diphosphate glucuronosyltransferase enzyme or UGT. A change of the UDP-glucuronosyltransferase gene – a faulty gene.
Gilbert’s Syndrome usually has no symptoms and can cause mild jaundice. Mild but prolonged jaundice can be a cause of worry as it increases the chances of serious liver disorders. The diagnostic plans for this disease include: Study of the medical history of the patient and the patient's family Physical examination of the liver Blood count test Liver biopsy Urine test Liver test
Gilbert’s Syndrome does not require major treatment, as it isn't a serious condition. Patients are known to have normal life expectancy and healthy lifestyles without treatment. In specific cases, however, jaundice can be managed by a balanced diet and adequate intake of water. If the symptoms of jaundice get too unsettling, patients may benefit from Phenobarbital, a medicine to lower bilirubin levels.
Gilbert’s Syndrome cannot be prevented as it is inherited, However, the following can help control the symptoms: Balanced diet Drink plenty of water Avoid alcohol Manage stress Avoid junk food
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